Suppression of Spry1 reduces HIF1α-dependent glycolysis and impairs angiogenesis in BRAF-mutant cutaneous melanoma.

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Tác giả: Eva Andreuzzi, Lorena Baboci, Arianna Bellazzo, Lucrezia Camicia, Francesca Colizzi, Marina Comelli, Alessia Covre, Michele Dal Bo, Albina Fejza, Elisabetta Fratta, Giorgio Giurato, Roberto Guerrieri, Jessica Lamberti, Michele Maio, Domenico Memoli, Maurizio Mongiat, Barbara Montico, Giovanni Nassa, Tuula A Nyman, Patrizia Sabatelli, Annamaria Salvati, Luca Sigalotti, Agostino Steffan, Alessandro Weisz

Ngôn ngữ: eng

Ký hiệu phân loại: 005.746 Data compression

Thông tin xuất bản: England : Journal of experimental & clinical cancer research : CR , 2025

Mô tả vật lý:

Bộ sưu tập: NCBI

ID: 105770

BACKGROUND: About 50% of cutaneous melanoma (CM) harbors the activating BRAF MATERIALS AND METHODS: Immunoprecipitation coupled to mass spectrometry was employed to gain insight into Spry1 interactome. Spry1 gene was knocked-out using the CRISPR strategy in the BRAF-mutant cell lines. Transmission electron microscopy was used to assess the relationship between Spry1 expression and mitochondrial morphology. By using in vitro and in vivo models, the effects of Spry1 RESULTS: Spry1 was mainly located in mitochondria in BRAF CONCLUSIONS: Altogether, these findings identify Spry1 as a potential regulator of mitochondrial homeostasis, and uncover a previously unrecognized role for Spry1 in regulating nuclear HIF1α expression and angiogenesis in BRAF SIGNIFICANCE: Spry1
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