Adipose tissue deficiency impairs transient lipid accumulation and delays liver regeneration following partial hepatectomy in male Seipin knockout mice.

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Tác giả: Xin Chen, Qianqian Dong, Mingming Gao, Chenxi Liang, Yang Liu, Ziwei Liu, Kexin Ma, Yidan Ma, Jinye Tang, Mengyu Wang, Xiaowei Wang, Xiaoqin Wu, Hongyuan Yang, Yaru Zhou

Ngôn ngữ: eng

Ký hiệu phân loại:

Thông tin xuất bản: United States : Clinical and translational medicine , 2025

Mô tả vật lý:

Bộ sưu tập: NCBI

ID: 236131

 BACKGROUND: Liver diseases pose significant health challenges, underscoring the importance of understanding liver regeneration mechanisms. Systemic adipose tissue is thought to be a primary source of lipids and energy during this process
  however, empirical data on the effects of adipose tissue deficiency are limited. This study investigates the role of adipose tissue in liver regeneration, focusing on transient regeneration-associated steatosis (TRAS) and hepatocyte proliferation using a Seipin knockout mouse model that mimics severe human lipodystrophy. Additionally, the study explores therapeutic strategies through adipose tissue transplantation. METHODS: Male Seipin knockout (Seipin RESULTS: Seipin CONCLUSIONS: Systemic adipose tissue is essential for TRAS and effective liver regeneration following PHx. Its deficiency impairs these processes, while adipose tissue transplantation can restore normal liver function. These findings underscore the critical role of adipose tissue in liver recovery and suggest potential therapeutic strategies for liver diseases associated with lipodystrophies. KEY POINTS: Seipin
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