Intestinal human carboxylesterase 2 (CES2) expression rescues drug metabolism and most metabolic syndrome phenotypes in global Ces2 cluster knockout mice.

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Tác giả: Jos H Beijnen, Joke Beukers-Korver, Rahmen Bin Ali, Chang-Pei Gan, Ivo Huijbers, Maria C Lebre, Wen-Long Li, Colin Pritchard, Hilde Rosing, Alfred H Schinkel, Ji-Ying Song, Els Wagenaar, Yao-Geng Wang

Ngôn ngữ: eng

Ký hiệu phân loại: 937.07 Early and middle periods, 31 B.C.–284 A.D.

Thông tin xuất bản: United States : Acta pharmacologica Sinica , 2025

Mô tả vật lý:

Bộ sưu tập: NCBI

ID: 251798

Carboxylesterase 2 (CES2) is expressed mainly in liver and intestine, but most abundantly in intestine. It hydrolyzes carboxylester, thioester, and amide bonds in many exogenous and endogenous compounds, including lipids. CES2 therefore not only plays an important role in the metabolism of many (pro-)drugs, toxins and pesticides, directly influencing pharmacology and toxicology in humans, but it is also involved in energy homeostasis, affecting lipid and glucose metabolism. In this study we investigated the pharmacological and physiological functions of CES2. We constructed Ces2 cluster knockout mice lacking all eight Ces2 genes (Ces2
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