Yttrium-90-Labeled Anti-Glypican 3 Radioimmunotherapy Halts Tumor Growth in an Orthotopic Xenograft Model of Hepatocellular Carcinoma [electronic resource]

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Tác giả:

Ngôn ngữ: eng

Ký hiệu phân loại: 616.99 Tumors and miscellaneous communicable diseases

Thông tin xuất bản: Washington, D.C. : Oak Ridge, Tenn. : United States. Dept. of Energy. Office of Science ; Distributed by the Office of Scientific and Technical Information, U.S. Dept. of Energy, 2019

Mô tả vật lý: Size: Article No. 4564707 : , digital, PDF file.

Bộ sưu tập: Metadata

ID: 259974

 Hepatocellular carcinoma (HCC) is the second most lethal malignancy globally and is increasing in incidence in the United States. Unfortunately, there are few effective systemic treatment options, particularly for disseminated disease. Glypican-3 (GPC3) is a proteoglycan cell surface receptor overexpressed in most HCCs and provides a unique target for molecular therapies. We have previously demonstrated that PET imaging using a <
 sup>
 89<
 /sup>
 Zr-conjugated monoclonal anti-GPC3 antibody (<
 em>
 ?<
 /em>
 GPC3) can bind to minute tumors and allow imaging with high sensitivity and specificity in an orthotopic xenograft mouse model of HCC and that serum alpha-fetoprotein (AFP) levels are highly correlated with tumor size in this model. In the present study, we conjugated <
 sup>
 90<
 /sup>
 Y, a high-energy beta-particle-emitting radionuclide, to our <
 em>
 ?<
 /em>
 GPC3 antibody to develop a novel antibody-directed radiotherapeutic approach for HCC. Luciferase-expressing HepG2 human hepatoblastoma cells were orthotopically implanted in the livers of athymic nude mice, and tumor establishment was verified at 6 weeks after implantation by bioluminescent imaging and serum AFP concentration. Tumor burden by bioluminescence and serum AFP concentration was highly correlated in our model. Yttrium-90 was conjugated to <
 em>
 ?<
 /em>
 GPC3 using the chelating agent 1,4,7,10-tetraazacyclododecane-1,4,7,10-tetraacetic acid (DOTA) and injected via the tail vein into the experimental mice at a dose of 200 <
 em>
 ?<
 /em>
 Ci/mouse or 300 <
 em>
 ?<
 /em>
 Ci/mouse. Control mice received DOTA-<
 em>
 ?<
 /em>
 GPC3 without radionuclide. At 30 days after a single dose of the radioimmunotherapy agent, mean serum AFP levels in control animals increased dramatically, while animals treated with 200 <
 em>
 ?<
 /em>
 Ci only experienced a minor increase, indicating cessation of tumor growth, and animals treated with 300 <
 em>
 ?<
 /em>
 Ci experienced a reduction in serum AFP concentration, indicating tumor shrinkage. Mean tumor-bearing liver weight in control animals was also significantly greater than that in animals that received either dose of <
 sup>
 90<
 /sup>
 Y-<
 em>
 ?<
 /em>
 GPC3. These results were achieved without significant toxicity as measured by body condition scoring and body weight. The results of this preclinical pilot demonstrate that GPC3 can be used as a target for radioimmunotherapy in an orthotopic mouse model of HCC and may be a target of clinical significance, particularly for disseminated HCC.
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