Interaction between APOE Ɛ4 status, chemotherapy and endocrine therapy on cognitive functioning among breast cancer survivors: the CANTO-Cog longitudinal study.

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Tác giả: Chayma Bousrih, Sandrine Boyault, Hélène Castel, François Christy, Dominique Delmas, Antonio Di Meglio, Mylène Duivon, Patricia A Ganz, Catherine Gaudin, Florence Joly, Marie Lange, Justine Lequesne, Florence Lerebours, Christelle Lévy, Olivier Rigal, Emilie Thomas, Kathleen Van Dyk, Ines Vaz Luis

Ngôn ngữ: eng

Ký hiệu phân loại: 004.6 Interfacing and communications

Thông tin xuất bản: England : Breast cancer research : BCR , 2025

Mô tả vật lý:

Bộ sưu tập: NCBI

ID: 469442

BACKGROUND: Apolipoprotein Ɛ4 genotype (APOE4) has been associated with cancer-related cognitive impairment, but its interaction with treatments remains unclear. This longitudinal study aims to evaluate the association between APOE4 and cognitive impairment in women with breast cancer (BC) undergoing chemotherapy (CT) or endocrine therapy (ET). FINDINGS: Patients with stage I-III breast cancer completed cognitive tests at diagnosis (before surgery), then at year-1, year-2, and year-4 post-diagnosis. APOE4 status (APOE4+ [carriers] vs. APOE4- [non-carriers]) was genotyped from blood sample. Cognitive outcomes included episodic memory, working memory, attention, processing speed, and executive functions. Patients were defined as having overall cognitive impairment if ≥ 2 domains were impaired. We fitted logistic and linear mixed models to assess associations of APOE4 status with cognitive impairment over time and interactions of APOE4 with CT and ET. Among 334 patients, 64 (19%) were APOE4+, 117 (35%) patients were treated with CT, 41 (12%) with ET, and 162 (49%) with CT+ET. There were no significant association between overall cognitive impairment and APOE4, nor interactions with CT or ET. At year-4, APOE4+ patients treated with ET had lower attention performance than APOE4- patients not treated with ET, and APOE4+ patients not treated with ET had lower episodic memory performance than APOE4- patients not treated with ET. CONCLUSIONS: This study suggests APOE4 genotyping is ineffective for detecting cognitive impairment in BC. New genotypes should be identified to predict cognitive decline in BC.
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