Đánh giá 44 vị trí tái hẹp trên 50%. Có 24 tổn thương tái cấu trúc âm tính. Nôi mạc tăng sinh gặp ở cả 44 vị trí, 31 vị trí tổn thương có tái hẹp tại rìa stent có hoặc không kèm theo tái hẹp trong thân stent. 12 vị trí stent méo hoặc không áp sát, 19 stent nhỏ hoặc nở không hết, 7 vị trí hẹp tại chỗ 2 stent gối nhau. Tái hẹp điểm gặp ở 23/44 vị trí.Previous researchs have shown combining IVUS image with coronary angiography to provides results with better acc-uracy and explanation for the mechanism of ISR. Results
Evaluating 44 ISR position >
50% (through coronary angiography). Stent implantation period is 46,02 ± 33,4 months. Restenosis percentage on IVUS: 67,7 ± 7,3%. 24 positions (63.2%) of negative remodeling injury were reported. Intimal hyperplasia was reported in all 44 cases. Restenosis with fibrosis plaque and mixed plaque was reported at 74%, 7 (15,9%) positions have plaque vulnerability and rupture. 31 (70.4%) positions were reported with Edge stent restenosis. 12 cases (27.2%) of malapposition or distorted stent, 19 cases (43,2%) in those stents were underexpansion or undersize, 7 cases (15,9%) of strut stent overlap were reported. Focal restenosis was reported at 23 (52,3%) positions. 22 (50%) positions have stent area under 9mml MLSA: 8,5 ± 2,8 (mm2) smaller Oian that of minimal lumen reference area: 9,5 ± 2,6 (mm2) (D=0,01). Conclusion: IVUS allows us to assess the characterization of plaque types, restenosis stent locations, restenosis morphology, phenomenon of vascular remodeling, and post-intervention stent conditions. In most IRS, IH was the dominant mechanism of ISR.