Reduction of Neuroinflammation as a Common Mechanism of Action of Anorexigenic and Orexigenic Peptide Analogues in the Triple Transgenic Mouse Model of Alzheimer´s Disease.

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Tác giả: Jaroslav Kuneš, Lenka Maletínská, Anna Mengr, Andrea Pačesová, Zuzana Šmotková, Blanka Železná

Ngôn ngữ: eng

Ký hiệu phân loại: 230.071 Education in Christianity, in Christian theology

Thông tin xuất bản: United States : Journal of neuroimmune pharmacology : the official journal of the Society on NeuroImmune Pharmacology , 2025

Mô tả vật lý:

Bộ sưu tập: NCBI

ID: 55107

Alzheimer's disease (AD) is the most common form of dementia. Characterized by progressive neurodegeneration, AD typically begins with mild cognitive decline escalating to severe impairment in communication and responsiveness. It primarily affects cerebral regions responsible for cognition, memory, and language processing, significantly impeding the functional independence of patients. With nearly 50 million dementia cases worldwide, a number expected to triple by 2050, the need for effective treatments is more urgent than ever. Recent insights into the association between obesity, type 2 diabetes mellitus, and neurodegenerative disorders have led to the development of promising treatments involving antidiabetic and anti-obesity agents. One such novel promising candidate for addressing AD pathology is a lipidized analogue of anorexigenic peptide called prolactin-releasing peptide (palm
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