SRSF12 deficiency enhances tumor innervation and accelerates pancreatic tumorigenesis.

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Tác giả: Zong-Hao Duan, Xue-Liang Fu, Rong Hua, Yan-Miao Huo, Qin-Yuan Jia, De-Jun Liu, Wei Liu, Ding Ma, Xue-Shi-Yu Ma, Yong-Wei Sun, Da-Peng Xu, Kan Xu, Yan-Nan Xu, Jian-Yu Yang, Min-Wei Yang, Yi-Fan Yin, Jun-Feng Zhang

Ngôn ngữ: eng

Ký hiệu phân loại: 620.1126 Engineering mechanics and materials

Thông tin xuất bản: Ireland : Cancer letters , 2025

Mô tả vật lý:

Bộ sưu tập: NCBI

ID: 552363

The peripheral nervous system significantly determines the fate of solid tumors and their microenvironment. In neurotropic malignancies such as pancreatic and prostate cancer, denervation in animal models demonstrate significantly delays in tumor initiation and progression, underscoring the critical neural dependency of these cancers. While tumor innervation establishes a structural basis for the neuromodulatory effects, the degree of innervation exhibits marked heterogeneity across tumor types, and its regulatory mechanisms remain poorly characterized. In this study, we screened genes associated with innervation status in pancreatic cancer and identified the splicing factor SRSF12 as a critical gene related to tumor innervation. In clinical samples, SRSF12 was expressed at low levels in pancreatic cancer tissues, and its downregulation was linked to poor prognosis in patients. Then we crossed Kras mutation and Srsf12 knockout mice (Kras
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