Nfil3 contributes to renal fibrosis by activating fibroblasts through directly promoting the expression of Spp1.

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Tác giả: Weijuan Gong, Huanan Li, Peifen Li, Qinhao Shen, Weigan Shen, Min Yang, Ming Yang, Xueying Zhang, Zifan Zhu

Ngôn ngữ: eng

Ký hiệu phân loại: 658.32259 Personnel management (Human resource management)

Thông tin xuất bản: Netherlands : Biochimica et biophysica acta. Molecular basis of disease , 2025

Mô tả vật lý:

Bộ sưu tập: NCBI

ID: 552455

 The activation of fibroblasts into myofibroblasts and the expansion of myofibroblasts are key processes contributing to renal fibrosis
  however, the precise underlying mechanisms remain largely unclear. In this study, we found that nuclear factor, interleukin 3 regulated (Nfil3), a basic leucine zipper transcription factor, was significantly upregulated in fibroblasts in kidney tissues from mouse models of unilateral ureteral obstruction (UUO)-induced renal fibrosis and kidney biopsies from patients with renal fibrosis. Conditional knockout of Nfil3 in fibroblasts (Nfil3
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