Overexpression of SHANK2 contributes malignant outcomes as a Hippo pathway regulator in gastric cancer.

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Tác giả: Hiroshi Arakawa, Tomohiro Arita, Hitoshi Fujiwara, Satoshi Hamada, Taisuke Imamura, Ryo Ishida, Hajime Kamiya, Jun Kiuchi, Shuhei Komatsu, Hirotaka Konishi, Takeshi Kubota, Keiji Nishibeppu, Takuma Ohashi, Eigo Otsuji, Hiroki Shimizu, Atsushi Shiozaki, Yusuke Takashima, Hitoshi Tsuda, Masateru Yamauchi

Ngôn ngữ: eng

Ký hiệu phân loại: 912.01 Philosophy and theory

Thông tin xuất bản: United States : American journal of cancer research , 2025

Mô tả vật lý:

Bộ sưu tập: NCBI

ID: 57190

BACKGROUND: Recent studies identified that SH3 and multiple ankyrin repeat domains 2 (SHANK2) is located in a gene-amplified region 11q13 of various human cancers, and has oncogenic functions as a Hippo pathway suppressor in hepatocellular carcinoma. In this study, we tested whether SHANK2 acts as a cancer-promoting gene through its activation or overexpression in gastric cancer (GC). MATERIALS AND METHODS: We analyzed 5 GC cell lines and 172 primary tumor samples of GC, which were curatively resected in our hospital. RESULTS: Overexpression of SHANK2 protein was frequently detected in GC cell lines (4/5 cell lines, 80%). Knockdown of SHANK2 inhibited cell proliferation, migration and invasion of GC cells in a CONCLUSIONS: These findings suggest that SHANK2 plays a crucial role in tumor malignant potential through the Hippo pathway suppression and highlight its usefulness as a prognostic factor and potential therapeutic target in GC.
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