Microbiota-derived 3-Methyl-L-histidine mediates the proatherogenic effect of high chicken protein diet.

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Tác giả: Jialin He, Wenkang Li, Ludi Liu, Yan Liu, Min Xia, Yingxi Xu, Bingqi Ye, Yawen Zhao, Jiangyuan Zhu, Shanshan Zhu

Ngôn ngữ: eng

Ký hiệu phân loại: 912.01 Philosophy and theory

Thông tin xuất bản: China : MedComm , 2025

Mô tả vật lý:

Bộ sưu tập: NCBI

ID: 57218

 Diet rich in chicken protein has gained a widespread popularity for its profound effect on weight loss and glycemic control
  however, its long-term effect on cardiovascular health and the underlying mechanisms remains obscure. Here, we demonstrated that higher intake of chicken protein was an independent risk factor for sub-clinical atherosclerosis. Adherence to high chicken protein diet (HCD) alleviated excessive weight gain and glycemic control regardless of the presence of gut microbiota in apolipoprotein E-deficient mice. In contrast, long-term HCD administration enhanced intestinal cholesterol absorption and accelerated atherosclerotic plaque formation in a gut microbiota-dependent manner. Integrative analysis of 16S rDNA sequencing and metabolomics profiling identified 3-Methyl-L-histidine (3-MH), resulting from an enrichment of
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