CCRL2 promotes the interferon-γ signaling response in myeloid neoplasms with erythroid differentiation and mutated

• Tác giả: Abdulmuez Abdulmalik, Alexander J Ambinder, Marios Arvanitis, Tatianna R Boronina, Frederick Bunz, Theodora Chatzilygeroudi, Robert N Cole, Amy E DeZern, Christopher Esteb, Priyanka Fernandes, Ivana Gojo, Lukasz Gondek, Yashvi Hemani, Richard J Jones, Styliani Karanika, Theodoros Karantanos, Mark J Levis, Chen Lossos, Leo Luznik, Nour Sabiha Naji, Sergiu Pasca, Suman Paul, Bogdan Paun, Brandy Perkins, Linda S Resar, Joseph Rimando, Ilias Sinanidis, Conover Talbot, Pablo Toledano-Sanz, Panagiotis Tsakiroglou, Rena Xian, Xinghan Zeng
• Ngôn ngữ: eng
• Ký hiệu phân loại: 297.1248 Sources of Islam
• Thông tin xuất bản: United States : bioRxiv : the preprint server for biology , 2025
• Bộ sưu tập: NCBI
• ID: 682824

Tóm tắt

UNLABELLED: Patients with myeloid neoplasms with loss-of-function ONE SENTENCE SUMMARY: CCRL2 is overexpressed in AML with loss-of-function