Role of RGS17 in cisplatin-induced cochlear inflammation and ototoxicity via caspase-3 activation.

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Tác giả: Raheem F H Al Aameri, Dheyaa Al Sallami, Entkhab M A Alanisi, Ian Alberts, Vickram Ramkumar, Leonard P Rybak, Shelley Tischkau

Ngôn ngữ: eng

Ký hiệu phân loại: 371.4046 Student guidance and counseling

Thông tin xuất bản: Switzerland : Frontiers in immunology , 2025

Mô tả vật lý:

Bộ sưu tập: NCBI

ID: 684078

Cisplatin is a chemotherapy drug used to treat different solid tumors, including ovarian, bladder, lung, and head and neck cancers. One of its significant side effects is ototoxicity, especially when high doses are required. Cisplatin-induced ototoxicity is associated with increased cochlear cell death resulting from DNA damage, caspase activation, oxidative stress, inflammation, and glutamate excitotoxicity. The regulator of G protein signaling 17 (RGS17), a member of the RGS-RZ subfamily, hastens the hydrolysis of GTP to GDP on the G
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