Activation of gut microbiota-hif1α axis effectively restores resistance to Aeromonas veronii caused by improper administration of AiiO-AIO6.

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Tác giả: Qianwen Ding, Le Luo Guan, Shenghui Li, Shengkang Li, Delong Meng, Benjamin Niemann, Chao Ran, Tsegay Teame, Tiantian Wang, Rui Xia, Xiaoqing Xu, Hongwei Yang, Yalin Yang, Yuanyuan Yao, Yaqing Zhang, Zhen Zhang, Zhigang Zhou

Ngôn ngữ: eng

Ký hiệu phân loại:

Thông tin xuất bản: United States : The Journal of nutrition , 2025

Mô tả vật lý:

Bộ sưu tập: NCBI

ID: 685621

BACKGROUND: Feeding adult zebrafish a diet supplemented with quenching enzyme AiiO-AIO6 for 3 wk improved the growth performance and disease resistance. However, when the feeding period was extended to 8 wk, zebrafish's disease resistance to Aeromonas veronii decreased. OBJECTIVE: We investigated the mechanisms of the reduced disease resistance of zebrafish induced by feeding on AiiO-AIO6 supplemented diet for a long-term (8 wk), and assessed the effectiveness of feed additives in restoring the low disease resistance. METHODS: One-month-old (adult) zebrafish were fed with a basal diet and the basal diet supplemented with AiiO-AIO6 (10 U/g) for 8 wk (Expt. 1). Furthermore, zebrafish larvae model (Expt. 2) was developed and used to study the mechanisms how AiiO-AIO6 affected the disease resistance (Expt. 3). We also investigated the effectiveness of selected prebiotic tributyrin, β-glucan or mannan in activating gut microbiota-hif1α to restore the low disease resistance of adult zebrafish fed with AiiO-AIO6 for 8 wk (Expt. 4). Lastly, the effects of Bacillus subtilis in activating the gut microbiota-hif1α and improving the low disease resistance of zebrafish larvae induced by AiiO-AIO6 was examined (Expt. 5). RESULTS: Feeding adult zebrafish with AiiO-AIO6 for 8 wk promoted the growth but disordered the gut microbiota and reduced disease resistance. Zebrafish larvae model confirmed that feeding AiiO-AIO6 for 2 d increased disease resistance, while 7 d decreased the resistance by suppressing hif1α. Using a germ-free zebrafish larvae model, we also demonstrated that AiiO-AIO6-induced gut microbiota mediated inhibition of hif1α. Furthermore, zebrafish fed on the AiiO-AIO6 containing diet supplement with tributyrin, β-glucan, mannan, or B. subtilis activated the gut microbiota-hif1α axis to reverse the low resistance caused by AiiO-AIO6. CONCLUSION: Activating the gut microbiota-hif1α axis has vital role in improving intestinal health and restores the low resistance to A. veronii caused by improper administration of dietary AiiO-AIO6 in zebrafish.
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