Bidirectional Mendelian randomization and mediation analysis of million-scale data reveal causal relationships between thyroid-related phenotypes, smoking, and lung cancer.

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Tác giả: Feng Chen, Jiajin Chen, Qiuyuan Chen, David C Christiani, Ruili Hou, Can Ju, Yi Li, Lijuan Lin, Aoxuan Wang, Xiang Wang, Xuan Wang, Yanru Wang, Yongyue Wei, Ning Xie, Xiaowen Xu, Ruyang Zhang, Zhongwen Zhang, Mengsheng Zhao

Ngôn ngữ: eng

Ký hiệu phân loại: 618.97703 Pediatrics and geriatrics

Thông tin xuất bản: China : Journal of biomedical research , 2025

Mô tả vật lý:

Bộ sưu tập: NCBI

ID: 686631

Emerging evidence highlights the role of thyroid hormones in cancer, though findings are controversial. Research on thyroid-related traits in lung carcinogenesis is limited. Using UK Biobank data, we conducted bidirectional Mendelian randomization (MR) to assess causal links between lung cancer risk and thyroid dysfunction (hypothyroidism/hyperthyroidism) or function traits (free thyroxine [FT4], normal-range TSH). Furthermore, in the smoking-behavior stratified MR analysis, we evaluated the mediating effect of thyroid-related phenotypes on the association between smoking phenotype and lung cancer. We confirmed significant associations between lung cancer risk and hypothyroidism (hazards ratio [HR] = 1.14, 95% confidence interval [CI] = 1.03-1.26,
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