Quercetin-3-Methyl Ether Induces Early Apoptosis to Overcome HRV1B Immune Evasion, Suppress Viral Replication, and Mitigate Inflammatory Pathogenesis.

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Tác giả: Won-Jin Baek, Jae-Hoon Chang, Sungchan Cho, Hwa-Jung Choi, Sun Shim Choi, Youngwook Ham, Dong-Yeop Kim, Min-Jung Kim, Seong-Ryeol Kim, Hyun-Jeong Ko, Yong Soo Kwon, Sunil Mishra, Seo-Hyeon Mun, Jae-Hyoung Song, Heejung Yang

Ngôn ngữ: eng

Ký hiệu phân loại: 939.401 Early history to ca. 1900 B.C.

Thông tin xuất bản: Korea (South) : Biomolecules & therapeutics , 2025

Mô tả vật lý:

Bộ sưu tập: NCBI

ID: 691569

Human rhinovirus (HRV) causes the common cold and exacerbates chronic respiratory diseases, such as asthma and chronic obstructive pulmonary disease. Despite its significant impact on public health, there are currently no approved vaccines or antiviral treatments for HRV infection. Apoptosis is the process through which cells eliminate themselves through the systematic activation of intrinsic death pathways in response to various stimuli. It plays an important role in viral infections and serves as a key immune defense mechanism in the interactions between viruses and the host. In the present study, we investigated the antiviral effects of quercetin-3-methyl ether, a flavonoid isolated from
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