Olfactory mucosal mesenchymal stem cell-derived exosome Lnc A2M-AS1 ameliorates oxidative stress by regulating TP53INP1-mediated mitochondrial autophagy through interacting with IGF2BP1 in Parkinson's diseases.

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Tác giả: Dan Hou, Chuang Wang, Ying Xia, Guoshuai Yang, Jiangshan Zhang, Yanhui Zhou

Ngôn ngữ: eng

Ký hiệu phân loại: 181.4 *India

Thông tin xuất bản: Switzerland : Cell biology and toxicology , 2025

Mô tả vật lý:

Bộ sưu tập: NCBI

ID: 725749

 BACKGROUND: Exosome Lnc A2M-AS1 from olfactory mucosa mesenchymal stem cells (OM-MSCs) can ameliorate oxidative stress by improving mitophagy in cardiomuscular cells
  however, it remains unclear whether this effect exists in the brain tissues of patients with Parkinson's disease (PD). METHODS: OM-MSC-Exosomes were isolated and verified based on morphology and specific biomarkers. The effects of OM-MSC-Exo on mitochondrial autophagy, oxidative stress, and lncRNA A2M-AS1 were detected in MPP RESULTS: Exosomes isolated from olfactory mucosa mesenchymal stem cells were found to be rich in Lnc A2M-AS1. Lnc A2M-AS1 was proved to be able to ameliorate oxidative stress induced by MPP CONCLUSION: Collectively, lncRNA A2M-AS1 from OM-MSC-derived exosomes regulates TP53INP1 expression by targeting IGF2BP1 to induce mitophagy and ameliorate oxidative stress. OM-MSC-derived exosomes could potentially serve as promising candidates for new treatment methods for PD.
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