Parental obesity predisposes male and female offspring to exacerbated cardiac dysfunction and increased mortality after myocardial infarction.

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Tác giả: Alexandre A da Silva, Xuemei Dai, Jussara M do Carmo, John E Hall, Emily C Ladnier, Xuan Li, Marilia C Mouro, Ana C M Omoto, Odecio E S Tosta, Zhen Wang

Ngôn ngữ: eng

Ký hiệu phân loại: 553.676 Talc

Thông tin xuất bản: United States : American journal of physiology. Heart and circulatory physiology , 2025

Mô tả vật lý:

Bộ sưu tập: NCBI

ID: 726185

 Acute myocardial infarction (MI) is a leading cause of death worldwide, accounting for >
 1 million deaths/year in the U.S. alone. Although parental obesity is a risk factor for offspring cardiovascular diseases, the impact of parental obesity on offspring outcomes after MI is unknown. This study examined if non-obese male and female offspring from obese Sprague-Dawley rat parents fed high fat diet (HFD-Offs, n=11-19/sex) are at greater risk of death and worse cardiac dysfunction after MI, compared to offspring from lean parents fed normal diet (ND-Offs, n=12-15/sex). All offspring were fed ND from weaning, and subjected to left descending coronary artery ligation at 12 weeks of age to induce MI. Survival rate 24 hrs post-MI was examined and cardiac function was measured by echocardiography and intraventricular catheterization with a Millar catheter on day 7 post-MI. Compared to ND-Offs, male and female HFD-Off exhibited increased ventricular fibrillation and reduced survival post-MI (male: 37% vs 80% and female: 55% vs. 83% for HFD-Offs and ND-Offs, respectively). In surviving rats, systolic dysfunction was more pronounced in male and female HFD-Offs compared to ND-Offs at day 7 post-MI, despite similar infarct size in all groups. We also found reductions in baseline O
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