Hepatic HKDC1 deletion alleviates western diet-induced MASH in mice.

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Tác giả: Irene Corona-Avila, María Dolores Frutos, Grace Guzman, Md Wasim Khan, Dhruvi Makhanasa, María Ángeles Núñez-Sánchez, Medha Priyadarshini, Bruno Ramos-Molina, Pratham Viral Shah, Kai Xu

Ngôn ngữ: eng

Ký hiệu phân loại:

Thông tin xuất bản: Netherlands : Biochimica et biophysica acta. Molecular basis of disease , 2025

Mô tả vật lý:

Bộ sưu tập: NCBI

ID: 733887

The global prevalence of Metabolic Dysfunction-Associated Steatohepatitis (MASH) has been rising sharply, closely mirroring the increasing rates of obesity and metabolic syndrome. MASH exhibits a strong sexual dimorphism where females are affected with more severe forms after menopause. Hexokinase domain-containing protein 1 (HKDC1) has recently been recognized for its role in liver diseases, where its expression is minimal under normal conditions but significantly increases in response to metabolic stressors like obesity and liver injury. This selective upregulation suggests HKDC1's potential specialization in hepatic glucose and lipid dysregulation, linking it closely to the progression of MASH. This study aims to clarify the role of HKDC1 in Western diet-induced MASH in female mice by examining its impact on hepatic glucose and lipid metabolism, offering insights into its potential as a therapeutic target and addressing the need for sex-specific research in liver disease. This study reveals that HKDC1 expression is elevated in obese women with MASH and correlates with liver pathology. In a mouse model, liver-specific HKDC1 knockout (HKDC1
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