State-dependent functional network connectivity alterations in post-stroke dementia with subcortical lesions.

 0 Người đánh giá. Xếp hạng trung bình 0

Tác giả: Huaying Cai, Guocan Han, Yun Jiang, Jiaao Liaocheng, Linhui Ni, Dan Wu, Qianwen Wu, Nan Zhang, Zhiyong Zhao

Ngôn ngữ: eng

Ký hiệu phân loại:

Thông tin xuất bản: United States : Journal of Alzheimer's disease : JAD , 2025

Mô tả vật lý:

Bộ sưu tập: NCBI

ID: 738836

 BACKGROUND: Recent studies have revealed disrupted dynamic functional network connectivity (dFNC) in stroke or dementia brains. However, it remains unclear how the dFNC was altered in post-stroke dementia (PSD). OBJECTIVE: This study aimed to explore PSD-specific alterations in the dFNC and their association with clinical assessments. METHODS: We included 19 normal controls (NC), 16 PSD, and 20 post-stroke non-dementia (PSND) patients who underwent resting-state functional magnetic resonance imaging scan. Independent component analysis, combined with a sliding-window approach, was employed to calculate dFNC. The cognitive performance was assessed by both Mini-Mental State Examination and MiniCog assessments, which were subsequently used for multiple regression analysis to investigate the relationships with dFNC. RESULTS: We identified 13 meaningful resting-state networks, and the dFNC among them derived four states exhibiting different connection patterns. In state III with strong connections within high-order networks, PSD and PSND both showed increased connectivity between visual network (VN) and high-order networks relative to NC
  in state I with weak connections among all networks, PSD showed weaker connectivity between default mode network (DMN) and executive control network and between VN and DMN compared to PSND. Moreover, the dFNC measures showed significant correlations with cognitive assessments of patients. CONCLUSIONS: Our findings suggest that PSD-specific functional network connectivity (FNC) alterations are state-dependent, and dFNC reveals a coexistence mechanism of functional impairment and compensation in large-scale brain networks, which is not observed in static FNC. This offers a new perspective to understand the neural mechanisms of PSD.
Tạo bộ sưu tập với mã QR

THƯ VIỆN - TRƯỜNG ĐẠI HỌC CÔNG NGHỆ TP.HCM

ĐT: (028) 36225755 | Email: tt.thuvien@hutech.edu.vn

Copyright @2024 THƯ VIỆN HUTECH