Responses to checkpoint inhibition in metastatic triple negative breast cancer driven by divergent myeloid phenotypes.

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Tác giả: Tyler Alban, Zahraa Al-Hilli, Timothy Chan, C Marcela Diaz-Montero, Ivan Juric, Jennifer S Ko, Alberto J Montero, Prerana Bangalore Parthasarathy, Paul G Pavicic, Jennifer Powers, Patricia A Rayman, Salendra Singh, Avia D Wilkerson

Ngôn ngữ: eng

Ký hiệu phân loại: 629.1344 Aerospace engineering

Thông tin xuất bản: England : Communications medicine , 2025

Mô tả vật lý:

Bộ sưu tập: NCBI

ID: 746353

BACKGROUND: Given the scarcity of effective therapeutic targets, metastatic triple negative breast cancer (mTNBC) has shorter survival times compared to other advanced breast cancer subtypes. Although chemo-immunotherapy with immune checkpoint inhibitors (ICIs) in PD-L1 METHODS: We employed single cell RNA sequencing (scRNAseq), single cell secretomics, and flow cytometry to identify transcriptomic and proteomic peripheral immune cell signatures associated with response and non-response to anti-PD-1/PD-L1 therapy and chemotherapy in mTNBC. RESULTS: Transcriptomic analysis reveal divergent transcriptional programming of CD33 CONCLUSION: Our findings highlight CD33
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