An exploratory analysis of bezisterim treatment associated with decreased biological age acceleration, and improved clinical measure and biomarker changes in mild-to-moderate probable Alzheimer's disease.

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Tác giả: Clarence Ahlem, Robert Brooke, Juozas Gordevicius, Hira Javaid, Penelope Markham, Daniel E Martin-Herranz, Stephen O'Quinn, Nily Osman, Joseph Palumbo, Christopher L Reading, Lisa Schmunk, Donald C Simonson, Marcia A Testa, Lixia Wang, Jiayan Yan, Harvey Yuan, Jeffrey Zhang

Ngôn ngữ: eng

Ký hiệu phân loại: 809.008 History and description with respect to kinds of persons

Thông tin xuất bản: Switzerland : Frontiers in neuroscience , 2025

Mô tả vật lý:

Bộ sưu tập: NCBI

ID: 749189

INTRODUCTION: Aging is the primary risk factor for sporadic Alzheimer's disease. Chronic low-grade inflammation associated with aging drives cognitive impairment through multiple mechanisms involving oxidative stress, insulin resistance, and dysregulation of metabolic, immunologic, and hematologic systems. METHODS: In a 7-month, randomized, double-blind, placebo-controlled trial (NCT04669028), we investigated the safety and activity of bezisterim, a first-in-class, oral, blood-brain barrier-permeable, anti-inflammatory agent on cognitive, molecular, biochemical, physiological, and biological aging parameters in a subset of 50 mild-to-moderate probable Alzheimer's disease participants. These participants had source-document-verified clinical measures and samples, and they completed the protocol. This study focuses on epigenetic, metabolic, biomarker, and cognitive measures in the exploratory biomarker population that completed the protocol. RESULTS: Bezisterim was associated with non-significant directional improvements in multiple measures of cognitive and functional performance compared to placebo, with correlations to biological age (determined by DNA methylation "clocks") and to metabolism, inflammation, and dementia biomarkers. In addition, clinical measures correlated with the extent of DNA methylation of certain cytosine-phosphate-guanine (CpG) sites in genes associated with metabolic inflammation and neurodegeneration. DISCUSSION: The results suggest the possible use of bezisterim to target the multifactorial processes underlying dementia. CLINICAL TRIAL REGISTRATION: https://clinicaltrials.gov/study/NCT04669028, Identifier: NCT04669028.
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