Maintenance therapy with the FMS-like tyrosine kinase 3 inhibitor gilteritinib in patients with FMS-like tyrosine kinase 3-internal tandem duplication acute myeloid leukemia: A phase 2 study.

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Tác giả: Caroline Chen, David Delgado, Stanley C Gill, Emmanuel Gyan, Nahla Hasabou, Wensheng He, Jason E Hill, Martin Jädersten, Gunnar Juliusson, Richard J Kelly, Kohmei Kubo, Mark D Minden, Alessandro Rambaldi, László Szerafin

Ngôn ngữ: eng

Ký hiệu phân loại:

Thông tin xuất bản: United States : Cancer , 2025

Mô tả vật lý:

Bộ sưu tập: NCBI

ID: 80701

 BACKGROUND: The GOSSAMER phase 2 study assessed the FMS-like tyrosine kinase 3 (FLT3) inhibitor gilteritinib as maintenance therapy in patients with FLT3-internal tandem duplication (FLT3-ITD) acute myeloid leukemia (AML) in first complete remission without previous hematopoietic stem cell transplantation (HSCT). METHODS: Patients had to be within 2 months of their last consolidation cycle and have completed the recommended number of cycles per local practice. FLT3 inhibitors were allowed only during induction and/or consolidation. The primary end point was relapse-free survival (RFS). Secondary end points included overall survival (OS), event-free survival, and measurable residual disease (MRD). RESULTS: In total, 98 patients were randomized (gilteritinib, n = 63
  placebo, n = 35). RFS was not significantly different between the arms (hazard ratio, 0.74
  95% confidence interval, 0.41-1.34
  p = .16). RFS rates for the gilteritinib and placebo arms were 68.5% and 55.3% at 1 year, 51.8% and 44.9% at 2 years, and 41.2% and 40.8% at 3 years, respectively. OS was not significantly different between the arms but may have been affected by subsequent AML therapies after discontinuation. In patients who received subsequent therapy (gilteritinib, 46.8%
  placebo, 60.0%), a higher percentage of placebo-treated (57.1%) versus gilteritinib-treated patients (27.6%) underwent HSCT. At the end of treatment, 96.4% of gilteritinib-treated and 85.7% of placebo-treated patients had undetectable MRD. Relapsed placebo-treated (86.7%) versus gilteritinib-treated patients (34.8%) had a greater FLT3 mutational burden. No new significant safety concerns were noted. CONCLUSIONS: The primary end point was not achieved
  however, an observed trend toward potential benefit was noted in patients with FLT3-ITD AML who had not undergone prior HSCT.
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